p pa pb pc pd pe pf pg ph pi pk pl pm pn po pp pr ps pt pu pv pw px py

Перевод: pertussis speek pertussis


[существительное]
коклюш [мед.]


Тезаурус:

  1. This G protein family can be divided into two, depending on whether or not they are sensitive to pertussis toxin.
  2. As this mGlu receptor is coupled through a pertussis toxin-sensitive G protein (see Box 1), it is significant that LTP is suppressed by pretreatment with pertussis toxin.
  3. Agonist occupancy of the formyl peptide receptor may lead to a transient, pertussis toxin-sensitive increase in the concentration of free subunits which is sufficiently high to stimulate PLC- 2.
  4. Preincubation with pertussis toxin prevented the enhancement of the gastric inhibitory polypeptide response by TPA, suggesting an involvement of an inhibitory guanine nucleotide regulatory subunit of the Gi class, but did not change the inhibition of histamine stimulation.
  5. The WHO's expanded programme of immunization (EPI), launched in 1974, had helped prevent an estimated 2,200,000 deaths yearly from measles, neonatal diseases, tetanus and pertussis, as well as some 360,000 cases of poliomyelitis.
  6. One of these supplementary messages promoted immunisation against six major diseases; diphtheria, pertussis, tetanus, poliomyelitis, measles and tuberculosis.
  7. Activation of PLC- 2 by subunits may be an important mechanism by which pertussis toxin-sensitive G proteins stimulate PLC.
  8. This effect of PYY seems to be mediated by inhibition of cholinergic neurotransmission through a pertussis toxin sensitive mechanism.
  9. It is appropriate for infants who suffer events to be investigated for the presence of respiratory pathogens, particularly Bordetella pertussis and respiratory syncytial virus.
  10. These membranes also contain high numbers of receptors for N- formyl methionine-containing chemotactic peptides, which are coupled to stimulation of PLC through the pertussis toxin-sensitive G proteins G i2 and/or G i3 (ref. 17).
  11. Thus, in granulocytes, stimulation of PLC- 2 by the free subunit is likely to be the mechanism by which the pertussis toxin-sensitive G proteins G i2 and/or G i3 stimulate PLC.
  12. Receptor-controlled PLC is often regulated by G proteins, which can be modified by pertussis toxin in some cells but not in others.

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