a
aa
ab
ac
ad
ae
af
ag
ah
ai
aj
ak
al
am
an
ao
ap
aq
ar
as
at
au
av
aw
ax
ay
az
Перевод: antibody
[существительное] антивещество; антитело [существительное]
Тезаурус:
- Despite the lack of axonal degeneration in the transected optic nerves of C57B1/6-WLD mice (which we confirmed by labelling with the RT97 monoclonal antineurofilament antibody), the decrease in mitotic cells after transection was not significantly different in these mice than in normal C57B1/6 mice (Fig. 1 c), making it unlikely that the loss of an axolemmal mitogen is the cause of the decrease in proliferation.
- Antibody epitopes and ligand binding sites
- Finally, as in the thymuses of wild-type littermates, about 1% of the cells bear TCR in the thymus of the TCR - mutant mice, as determined by the anti-TCR- monoclonal antibody 3A10 (ref. 3) (Fig. 3b).
- The nature of the abnormality varied with the amount of antibody injected.
- Class I molecules were then immunoprecipitated with monoclonal antibody W6/32, which only recognizes class I molecules associated with 2 -microglobulin.
- Similar results were obtained when a rabbit anti-ribophorin I antiserum or immunoadsorbant-purified rat IgG2a monoclonal antibodies against the KDEL tetra- peptide were used to label the ER in double-labelling experiments with anti-Bcl-2 antibody (not shown).
- Note that the nuclear envelope, ER and mitochondria are labelled by the anti-Bcl-2 antibody.
- Embryos were stained with an anti-horseradish peroxidase antibody (Cappell), which recognizes a cell-surface antigen on all Drosophila neurons.
- Left, SDS gel stained with Coomassie blue (CB); middle, western blot labelled with an anti-63K channel monoclonal antibody (PMc 1D1) and an anti-240K monoclonal antibody (PMs 5E11).
- The small arrowhead indicates the unaffected rhombomere, ro2, as marked by axon staining with an anti-acetylated -tubulin antibody (kindly provided by G.Piperno).
- Some studies have shown that the Pill decreases antibody production slightly, and others have shown more allergies, infections and autoimmune diseases among Pill-takers.
- When, however, the cells were double-labelled with the anti-Bcl-2 antibody and either human autoantibodies against a set of mitochondrial proteins or a rabbit antiserum against ER proteins, it could be seen that Bcl-2 was present in the mitochondria (Fig. 3e, f), as well as in the nuclear envelope and ER (Fig. 3c, d).
- Dashed line represents the binding site of the monoclonal antibody 18A10, which can inhibit the calcium-mobilizing action of InsP 3 (ref.35).
|